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Produktbild: Molecular Pathology of Gynecologic Cancer
- 11%

Molecular Pathology of Gynecologic Cancer

Aus der Reihe Current Clinical Oncology
11% sparen

106,99 € UVP 120,99 €

inkl. gesetzl. MwSt., Versandkostenfrei

Beschreibung

Produktdetails

Einband

Taschenbuch

Erscheinungsdatum

23.08.2016

Herausgeber

Antonio Giordano + weitere

Verlag

Humana Press

Seitenzahl

226

Maße (L/B/H)

25,4/17,8/1,4 cm

Gewicht

478 g

Auflage

Softcover reprint of the original 1st ed. 2007

Sprache

Englisch

ISBN

978-1-4939-5677-7

Beschreibung

Rezension

"...a complete compilation of the molecular events leading to gynecologic cancers.  There are no other books that compete with it or address the issues it does." -Doody's Book Review, Weighted Numerical Score:90 - 4 Stars

Produktdetails

Einband

Taschenbuch

Erscheinungsdatum

23.08.2016

Herausgeber

Verlag

Humana Press

Seitenzahl

226

Maße (L/B/H)

25,4/17,8/1,4 cm

Gewicht

478 g

Auflage

Softcover reprint of the original 1st ed. 2007

Sprache

Englisch

ISBN

978-1-4939-5677-7

Herstelleradresse

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  • Produktbild: Molecular Pathology of Gynecologic Cancer
  • ***completed

    ________________________________________________________________________________
    MOLECULAR PATHOLOGY OF GYNECOLOGIC CANCER

    EDITORS : ANTONIO GIORDANO , M.D., Ph.D.
    ALESSANDRO BOVICELLI , M.D., Ph.D.
    ROBERT J. KURMAN, M.D., Ph.D.

    ***PREFACE
    Alessandro Bovicelli and Giovan Giacomo Giordano, Honorary President of the Italian Tumor Society (SIT)

    PART I : INTRODUCTION

    ***Chapter 1 : The Cell Cycle and the Molecular Biology of Cancer
    Giuseppina D'Andrilli, Alessandro Bovicelli and Antonio Giordano
    Sbarro Institute for Cancer Research and Molecular Medicine, Temple University, BioLife Science Building, Suite 333, 1900 N 12th Street. Philadelphia, PA, 19122.

    This chapter evaluates the molecules that conduct progression through the cell cycle and describes their activities that are linked together to control the process of cell reproduction. In light of these concepts, we believe that cancer is mainly a disease of the cell cycle based on the deregulation of this physiological process. This hypothesis is further elaborated upon and illustrated in the subsequent chapters .

    PART II : OVARIAN CANCER :

    ***Chapter 2 : Ovarian Serous Carcinogenesis-A Proposed Model
    Ie-Ming Shih and Robert. J Kurman
    Department of Pathology, The Johns Hopkins University School of Medicine, 401 North Broadway, Baltimore, Maryland, 21231-2410, USA.

    This chapter discusses the most innovative pathological issues that define ovarian neoplasia. The emphasis is placed on different ovarian pathways of carcinogenesis and progression to advanced disease. Particular focus is directed to borderline disease and its classification.

    ***Chapter 3 : Molecular Markers in Epithelial Ovarian Cancer.
    Pat Morin
    Laboratory of Cellular and Molecular Biology, Gerontology Research Center, NationalInstitute on Aging, Building 31, Room 5C27, 31 Center Drive, MSC 2292, Bethesda, MD 20892, morinp@grc.nia.nih.gov

    This chapter summarizes the studies which have correlated cancer screening, diagnosis and prognosis with structure and expression profiles of molecular markers. New insights made possible by the utilization of new technologies are discussed. The emphasis is placed on cell cycle regulatory genes . The diversity of cancer types and the value of examining multi-gene profiles is discussed in this chapter.

    PART III : ENDOMETRIAL CANCER :

    ***Chapter 4 : Introduction to Endometrial Cancer.
    Richard Zaino
    Department of Pathology, Milton S. Hershey Medical Center of Pennsylvania State University, Hershey, Pennsylvania 17033, USA

    This chapter discusses the most innovative pathological issues that define endometrial neoplasia . Special focus is placed on pre-neoplastic lesions. The criteria established for distinguishing lesions on the basis of architectural and cytological abnormalities are examined. Differences between simple or complex hyperplasia with or without atypia are also stressed. Histological types of advanced disease and implications for a different course of the disease are examined.

    ***Chapter 5 : Endometrial Carcinogenesis : An Integrated, Molecular, Histologic and Functional Model of a Dualistic Disease.
    Mutter G
    Pathology Institute, Brigham and Women's Hospital, 02115 Boston, Massachusetts.
    gmutter@rics.bwh.harvard.edu

    Among the hormone-related carcinomas, the best understood etiologically is endometrial cancer. The demographic characteristics of the disease as well as the non-demographic risk factors are mainly due to a cumulative exposure of the endometrium to that fraction of estrogen that is unopposed by the influence of progesterone. On the other hand, recent data in the literature defined two distinct pathways of endometrial carcinogenesis an